Ns [26,32,42-45]. However, the deleterious effects of AT1R antagonists in

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Ns [26,32,42-45]. However, the deleterious effects of AT1R antagonists in pregnancy [46,47] preclude their use for identifying the main receptor stimulated by excess angiotensin. The acute effect of angiotensin II plus a B2R blocker provoked an increase of systolic blood pressure in a subset of the group (responders) to values that were not attained with single interventions and in non-pregnant females receiving the combined treatment. The responders are probably bradykinin-dependent when circulating angiotensin II surpasses the endogenous gestational levels. This study leaves unresolved questions regarding why additive effects of combined interventions were observed for some, but not all, s.Ns [26,32,42-45]. However, the deleterious effects of AT1R antagonists in pregnancy [46,47] preclude their use for identifying the main receptor stimulated by excess angiotensin. The acute effect of angiotensin II plus a B2R blocker provoked an increase of systolic blood pressure in a subset of the group (responders) to values that were not attained with single interventions and in non-pregnant females receiving the combined treatment. The responders are probably bradykinin-dependent when circulating angiotensin II surpasses the endogenous gestational levels. This study leaves unresolved questions regarding why additive effects of combined interventions were observed for some, but not all, s.